Falkiewicz B, H. Brandt B, P. Bielawski K, Vogt U. Anomalies of ErbB (HER) receptor functions: mechanisms and methods of detection. Part I. Gene amplification of ErbB (HER) family. Contemporary Oncology/Współczesna Onkologia. 2000;4(3):102-107.
APA
Falkiewicz, B., H. Brandt, B., P. Bielawski, K., & Vogt, U. (2000). Anomalies of ErbB (HER) receptor functions: mechanisms and methods of detection. Part I. Gene amplification of ErbB (HER) family. Contemporary Oncology/Współczesna Onkologia, 4(3), 102-107.
Chicago
Falkiewicz, Bogdan, Burkhard H. Brandt, Krzysztof P. Bielawski, and Ulf Vogt. 2000. "Anomalies of ErbB (HER) receptor functions: mechanisms and methods of detection. Part I. Gene amplification of ErbB (HER) family". Contemporary Oncology/Współczesna Onkologia 4 (3): 102-107.
Harvard
Falkiewicz, B., H. Brandt, B., P. Bielawski, K., and Vogt, U. (2000). Anomalies of ErbB (HER) receptor functions: mechanisms and methods of detection. Part I. Gene amplification of ErbB (HER) family. Contemporary Oncology/Współczesna Onkologia, 4(3), pp.102-107.
MLA
Falkiewicz, Bogdan et al. "Anomalies of ErbB (HER) receptor functions: mechanisms and methods of detection. Part I. Gene amplification of ErbB (HER) family." Contemporary Oncology/Współczesna Onkologia, vol. 4, no. 3, 2000, pp. 102-107.
Vancouver
Falkiewicz B, H. Brandt B, P. Bielawski K, Vogt U. Anomalies of ErbB (HER) receptor functions: mechanisms and methods of detection. Part I. Gene amplification of ErbB (HER) family. Contemporary Oncology/Współczesna Onkologia. 2000;4(3):102-107.
The paper reviews mechanisms (gene amplifications, point mutations and deletions, gene expression alterations) leading to excessive activity of the ErbB (HER) family receptor tyrosine kinases, as well as techniques used in the research on these anomalies. Part I shortly describes the phenomenon of ErbB (HER) family gene amplification and methods used for its determination.
