Obtulowicz P, Stobiecki M, Dyga W, Popiela T, Obtulowicz K. Abdominal attack in a patient with hereditary angioedema due to C1 inhibitor deficiency complicated by a perforated peptic ulcer. Alergologia Polska - Polish Journal of Allergology. 2024;11(1):80-83. doi:10.5114/pja.2024.135548.
APA
Obtulowicz, P., Stobiecki, M., Dyga, W., Popiela, T., & Obtulowicz, K. (2024). Abdominal attack in a patient with hereditary angioedema due to C1 inhibitor deficiency complicated by a perforated peptic ulcer. Alergologia Polska - Polish Journal of Allergology, 11(1), 80-83. https://doi.org/10.5114/pja.2024.135548
Chicago
Obtulowicz, Piotr, Marcin Stobiecki, Wojciech Dyga, Tadeusz Popiela, and Krystyna Obtulowicz. 2024. "Abdominal attack in a patient with hereditary angioedema due to C1 inhibitor deficiency complicated by a perforated peptic ulcer". Alergologia Polska - Polish Journal of Allergology 11 (1): 80-83. doi:10.5114/pja.2024.135548.
Harvard
Obtulowicz, P., Stobiecki, M., Dyga, W., Popiela, T., and Obtulowicz, K. (2024). Abdominal attack in a patient with hereditary angioedema due to C1 inhibitor deficiency complicated by a perforated peptic ulcer. Alergologia Polska - Polish Journal of Allergology, 11(1), pp.80-83. https://doi.org/10.5114/pja.2024.135548
MLA
Obtulowicz, Piotr et al. "Abdominal attack in a patient with hereditary angioedema due to C1 inhibitor deficiency complicated by a perforated peptic ulcer." Alergologia Polska - Polish Journal of Allergology, vol. 11, no. 1, 2024, pp. 80-83. doi:10.5114/pja.2024.135548.
Vancouver
Obtulowicz P, Stobiecki M, Dyga W, Popiela T, Obtulowicz K. Abdominal attack in a patient with hereditary angioedema due to C1 inhibitor deficiency complicated by a perforated peptic ulcer. Alergologia Polska - Polish Journal of Allergology. 2024;11(1):80-83. doi:10.5114/pja.2024.135548.
A 30-year-old male with hereditary angioedema (HAE) due to C1 inhibitor deficiency was admitted to the Emergency Department (ED) due to severe abdominal pain lasting for 3 hs with severe bloating and weakness. pdC1INH and fluids were applied as usual and the patient was discharged with feeling better. A few hours later abdominal symptoms recurred and erythema appeared, thus the allergy to antispasmodic drug taken by the patient was suspected. In the ED, the patient received anti-allergic treatment and was discharged again. After 3 h severe abdominal symptoms returned with fever. The patient was readmitted to the ED with peritoneal symptoms. Abdominal X-ray confirmed digestive tract perforation. Laparotomy revealed perforation of the chronic duodenal ulcer. In conclusion, we indicate that a HAE patient with an abdominal pain not resolving after usually effective treatment, should be hospitalized and monitored. Differential diagnosis of reasons other than HAE attack should be made.
