Abstract
3/2012
vol. 29
Original paper
durEFFECTS
OF BLOOD LACTATE ON OXYGEN UPTAKE KINETICS DURING RECOVERY AFTER SPRINT IN HUMANS
Biol. Sport 2012;29:171-176
Online publish date: 2014/08/22
The purpose of this study was to examine the effects of blood lactate level (La) on oxygen uptake ( ·
VO2) kinetics during recovery after short-term exercise with maximal effort (sprint). Three sprints were performed on a cycle ergometer with a load of 8% of body weight at maximal rotation rate. ·
VO2 kinetics and oxygen debt were determined after three sprint tests: one 10-s cycling sprint, five repeated 10-s cycling sprints with 6-min intervals and one 30-s cycling sprint. There was no significant difference between peak power outputs in the 10-s sprint and five sprints. There was no difference in ·
VO2 kinetics during recovery from one sprint and during recovery after five sprints. La peaked at 5 min. The peak value of La was significantly lower in one sprint (4.41 ± 0.9 mM) than in five sprints (7.01 ± 2.2 mM). Thus, despite a difference in La, there was no difference between ·
VO2 kinetics during recovery after one sprint and after five sprints. There was a significant difference in ·
VO2 between the five sprints and 30-s sprint from 70 s to 320 s during recovery, but there were no significant differences in La after
5 min of recovery. There were two phases in ·
VO2. They consisted of fast oxygen debt and slow oxygen debt. There were also no differences in slow and fast oxygen debts between the two 10-s sprints despite significant differences in blood lactate during recovery. Peak La in the five sprints was not significantly different from that in the 30-s sprint (8.68 ± 1.2 mM). However, slow oxygen debt was significantly greater in the 30-s sprint than in the five sprints. It is concluded that ·
VO2 kinetics during recovery are not affected by an increase in blood lactate.
VO2) kinetics during recovery after short-term exercise with maximal effort (sprint). Three sprints were performed on a cycle ergometer with a load of 8% of body weight at maximal rotation rate. ·
VO2 kinetics and oxygen debt were determined after three sprint tests: one 10-s cycling sprint, five repeated 10-s cycling sprints with 6-min intervals and one 30-s cycling sprint. There was no significant difference between peak power outputs in the 10-s sprint and five sprints. There was no difference in ·
VO2 kinetics during recovery from one sprint and during recovery after five sprints. La peaked at 5 min. The peak value of La was significantly lower in one sprint (4.41 ± 0.9 mM) than in five sprints (7.01 ± 2.2 mM). Thus, despite a difference in La, there was no difference between ·
VO2 kinetics during recovery after one sprint and after five sprints. There was a significant difference in ·
VO2 between the five sprints and 30-s sprint from 70 s to 320 s during recovery, but there were no significant differences in La after
5 min of recovery. There were two phases in ·
VO2. They consisted of fast oxygen debt and slow oxygen debt. There were also no differences in slow and fast oxygen debts between the two 10-s sprints despite significant differences in blood lactate during recovery. Peak La in the five sprints was not significantly different from that in the 30-s sprint (8.68 ± 1.2 mM). However, slow oxygen debt was significantly greater in the 30-s sprint than in the five sprints. It is concluded that ·
VO2 kinetics during recovery are not affected by an increase in blood lactate.
Keywords
blood lactate, oxygen uptake kinetics, oxygen debt, cycling sprint, muscle fatigue
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