1. Samotij D, Nedoszytko B, Bartosińska J, et al. Pathogenesis of psoriasis in the “omic” era. Part I. Epidemiology, clinical manifestation, immunological and neuroendocrine disturbances. Adv Dermatol Allergol 2020; 37: 135-53.
2.
Hsu D, Fung M, Chen HL. Role of skin and gut microbiota in the pathogenesis of psoriasis, an inflammatory skin disease. Med Microecol 2020; 4 (Supl 1): 100016.
3.
Gisondi P, Bellinato F, Girolomoni G, Albanesi C. Pathogenesis of chronic plaque psoriasis and its intersection with cardiometabolic comorbidities. Front Pharmacol 2020; 11: 117.
4.
Dascǎlu R, Bărbulescu A, Stoica L, et al. Review: a contemporary, multifaced insight into psoriasis pathogenesis. J Personal Med 2024; 14: 535.
5.
Rønholt K, Nielsen AL, Johansen C, et al. IL-37 expression is downregulated in lesional psoriasis skin. Immunohorizons 2020; 4: 754-61.
6.
Wulamujiang A, Ozkanlı S, Coskunpinar E, et al. Lowered IL‐37 gene expression and elevated IL‐37‐producing tissue‐resident immune cells in psoriasis lesional biopsies. Pathol Int 2023; 73: 490-6.
7.
Watanabe A, Kamata M, Shimizu T, et al. Serum levels of angiogenesis‐related factors in patients with psoriasis. J Dermatol 2022; 50: 222-8.
8.
Conti P, Pregliasco F, Bellomo R, et al. Mast cell cytokines IL-1, IL-33, and IL-36 mediate skin inflammation in psoriasis: a novel therapeutic approach with the anti-inflammatory cytokines IL-37, IL-38, and IL-1Ra. Int J Mol Sci 2021; 22: 8076.
9.
Suzuki T, Hirakawa S, Shimauchi T, et al. VEGF-A promotes IL-17A-producing γδ T cell accumulation in mouse skin and serves as a chemotactic factor for plasmacytoid dendritic cells. J Dermatol Sci 2014; 74: 116-24.
10.
Chen Y, Tai Z, Zhu C, et al. Vascular endothelial growth factor A VEGFA inhibition: an effective treatment strategy for psoriasis. Int J Mol Sci 2024; 25: 59.
11.
Rendon A, Schäkel K. Psoriasis pathogenesis and treatment. Int J Mol Sci 2019; 20: 1475.
12.
Patil D, Sumathy TK, Shyamprasad AL. Relevance of serum vascular endothelial growth factor (VEGF) and serum interleukin-10 in the severity of psoriasis in psoriasis in South Indian patients. A case-control study. Turk J Dermatol 2023; 17: 6-10.
13.
Lao J, Xie Z, Qin Q, et al. Serum LL‐37and inflammatory cytokines levels in psoriasis. Immun Inflamm Dis 2024; 11: e802.
14.
Dombrowski Y, Schauber J. Cathelicidin LL‐37: a defense molecule with a potential role in psoriasis pathogenesis. Exp Dermatol 2012; 21: 327‐30.
15.
Nestle FO Kaplan DH, Bake J. Psoriasis. N Engl J Med 2009; 361: 496-509.
16.
Słucznowska-Głabowska S, Jaworska W, Staniszewska M, et al. Plasma levels of interleukins 36α, 36β, and 37 in patients with psoriasis and their correlation with disease activity parameters. J Clin Med 2022; 11: 5254.
17.
Yuan Y, Qiu J, Lin ZT, et al. Identification of novel autoantibodies associated with psoriatic arthritis. Arthritis Rheum 2019; 71: 941‐51.
18.
Tsuji G, Yamamura K, Kawamura K, et al. Regulatory mechanism of the IL-33–IL-37 axis via aryl hydrocarbon receptor in atopic dermatitis and psoriasis. Int J Mol Sci 2023; 24: 14633.
19.
Rønholt K, Nielsen AL, Johansen C, et al. IL-37 expression is downregulated in lesional psoriasis skin. Immunohorizons 2020; 4: 754-61.
20.
Luengas-Martinez A, Paus R, Young H. A novel personalized treatment approach for psoriasis: anti-VEGF-A therapy. Br J Dermatol 2023; 186: 782-91.
21.
Mohta A, Mohta A, Ghiya B. Assessing the association between psoriasis and cardiovascular ischemia: an investigation of vascular endothelial growth factor, cutaneous angiogenesis, and arterial stiffness. Indian Dermatol Online J 2023; 14: 653-7.
22.
Sobolev V, Muminova M, Zhukova O, et al. HGF, HNRPD, and sFLT1 interfere with the induction of VEGF in patients with severe psoriasis. Curr Mol Med 2024; 30: 477-89.
23.
Du Y, Jiang S, Cheng L, Liu J. JAK/STAT and VEGF/PAK1 signaling as emerging targets for topical treatment of psoriasis: a pilot study. Int J Clin Exp Pathol 2020; 13: 3111-9.
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